Advancing the Understanding of Vesicle-Associated Membrane Protein 1-Related Congenital Myasthenic Syndrome: Phenotypic Insights, Favorable Response to 3,4-Diaminopyridine, and Clinical Characterization of Five New Cases


Por: Natera-de Benito D, Pugliese A, Polavarapu K, Guergueltcheva V, Tournev I, Todorova A, Afonso Ribeiro J, Fernández-Mayoralas DM, Ortez-Gonzalez CI, Martorell-Sampol L, Estévez-Arias B, Matalonga L, Laurie S, Jou-Munoz C, Lau J, Thompson R, Shen X, Engel AG, Nascimento-Osorio A, Lochmüller H and Selcen D

Publicada: 1 ago 2024 Ahead of Print: 1 jun 2024
Resumen:
Background: Congenital myasthenic syndromes (CMS) are a group of inherited neuromuscular junction (NMJ) disorders arising from gene variants encoding diverse NMJ proteins. Recently, the VAMP1 gene, responsible for encoding the vesicle -associated membrane protein 1 (VAMP1), has been associated with CMS. Methods: This study presents a characterization of five new individuals with VAMP1-related CMS, providing insights into the phenotype. Results: The individuals with VAMP1-related CMS exhibited early disease onset, presenting symptoms prenatally or during the neonatal period, alongside severe respiratory involvement and feeding difficulties. Generalized weakness at birth was a common feature, and none of the individuals achieved independent walking ability. Notably, all cases exhibited scoliosis. The clinical course remained stable, without typical exacerbations seen in other CMS types. The response to anticholinesterase inhibitors and salbutamol was only partial, but the addition of 3,4-diaminopyridine (3,4 -DAP) led to significant and substantial improvements, suggesting therapeutic benefits of 3,4 -DAP for managing VAMP1-related CMS symptoms. Noteworthy is the identification of the VAMP1 (NM_014231.5): c.340delA; p.Ile114SerfsTer72 as a founder variant in the Iberian Peninsula and Latin America. Conclusions: This study contributes valuable insights into VAMP1-related CMS, emphasizing their early onset, arthrogryposis, facial and generalized weakness, respiratory involvement, and feeding difficulties. Furthermore, the potential efficacy of 3,4 -DAP as a useful therapeutic option warrants further exploration. The findings have implications for clinical management and genetic counseling in affected individuals. Additional research is necessary to elucidate the long-term outcomes of VAMP1-related CMS. (c) 2024 Elsevier Inc. All rights reserved.

Filiaciones:
Natera-de Benito D:
 Neuromuscular Unit, Department of Neurology, Hospital Sant Joan de Déu, Barcelona, Spain

 Applied Research in Neuromuscular Diseases, Institut de Recerca Sant Joan de Déu, Barcelona, Spain

Pugliese A:
 IRCCS Centro Neurolesi "Bonino-Pulejo", Neurology Unit, Messina, Italy

Polavarapu K:
 Children' s Hospital of Eastern Ontario Research Institute, Ottawa, Ontario, Canada

Guergueltcheva V:
 Clinic of Neurology, University Hospital Sofiamed, Sofia University St. Kliment Ohridski, Sofia, Bulgaria

Tournev I:
 Department of Neurology, University Hospital "Alexandrovska", Medical University, Sofia, Bulgaria

 Department of Cognitive Science and Psychology, New Bulgarian University, Sofia, Bulgaria

Todorova A:
 Genetic Medico-Diagnostic Laboratory "Genica", Sofia, Bulgaria

 Department of Medical Chemistry and Biochemistry, Sofia Medical University, Sofia, Bulgaria

Afonso Ribeiro J:
 Neurology Department, Coimbra University Hospital Centre, Coimbra, Portugal

Fernández-Mayoralas DM:
 Department of Pediatric Neurology, Hospital Universitario Quirónsalud, Madrid, Spain

Ortez-Gonzalez CI:
 Neuromuscular Unit, Department of Neurology, Hospital Sant Joan de Déu, Barcelona, Spain

 Applied Research in Neuromuscular Diseases, Institut de Recerca Sant Joan de Déu, Barcelona, Spain

Martorell-Sampol L:
 Department of Genetic and Molecular Medicine-IPER, Hospital Sant Joan de Déu and Institut de Recerca Sant Joan de Déu, Barcelona, Spain

Estévez-Arias B:
 Neuromuscular Unit, Department of Neurology, Hospital Sant Joan de Déu, Barcelona, Spain

 Laboratory of Neurogenetics and Molecular Medicine-IPER, Sant Joan de Deu Research Institute, Barcelona, Spain

Matalonga L:
 Centro Nacional de Análisis Genómico, Barcelona, Spain

Laurie S:
 Centro Nacional de Análisis Genómico, Barcelona, Spain

Jou-Munoz C:
 Applied Research in Neuromuscular Diseases, Institut de Recerca Sant Joan de Déu, Barcelona, Spain

 Department of Pathology, Hospital Sant Joan de Déu, Barcelona, Spain

Lau J:
 Children' s Hospital of Eastern Ontario Research Institute, Ottawa, Ontario, Canada

Thompson R:
 Children' s Hospital of Eastern Ontario Research Institute, Ottawa, Ontario, Canada

Shen X:
 Department of Neurology and Neuromuscular Research Laboratory, Mayo Clinic, Rochester, Minnesota

Engel AG:
 Department of Neurology and Neuromuscular Research Laboratory, Mayo Clinic, Rochester, Minnesota

Nascimento-Osorio A:
 Neuromuscular Unit, Department of Neurology, Hospital Sant Joan de Déu, Barcelona, Spain

 Applied Research in Neuromuscular Diseases, Institut de Recerca Sant Joan de Déu, Barcelona, Spain

Lochmüller H:
 Children' s Hospital of Eastern Ontario Research Institute, Ottawa, Ontario, Canada

 Laboratory of Neurogenetics and Molecular Medicine-IPER, Sant Joan de Deu Research Institute, Barcelona, Spain

 Brain and Mind Research Institute, University of Ottawa, Ottawa, Canada

 Department of Neuropediatrics and Muscle Disorders, Medical Center - University of Freiburg, Faculty of Medicine, Freiburg, Germany

Selcen D:
 Department of Neurology and Neuromuscular Research Laboratory, Mayo Clinic, Rochester, Minnesota
ISSN: 08878994





PEDIATRIC NEUROLOGY
Editorial
ELSEVIER SCIENCE INC, STE 800, 230 PARK AVE, NEW YORK, NY 10169, Estados Unidos America
Tipo de documento: Article
Volumen: 157 Número:
Páginas: 5-13
WOS Id: 001257474900001
ID de PubMed: 38833907
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