PPARß/d upregulates the insulin receptor ß subunit in skeletal muscle by reducing lysosomal activity and EphB4 levels


Por: Wang JR, Jurado-Aguilar J, Barroso E, Rodríguez-Calvo R, Camins A, Wahli W, Palomer FX and Vazquez M

Publicada: 18 dic 2024 Ahead of Print: 18 dic 2024
Resumen:
BackgroundThe increased degradation of the insulin receptor beta subunit (InsR beta) in lysosomes contributes to the development of insulin resistance and type 2 diabetes mellitus. Endoplasmic reticulum (ER) stress contributes to insulin resistance through several mechanisms, including the reduction of InsR beta levels. Here, we examined how peroxisome proliferator-activated receptor (PPAR)beta/delta regulates InsR beta levels in mouse skeletal muscle and C2C12 myotubes exposed to the ER stressor tunicamycin.MethodsWild-type (WT) and Ppard-/- mice, WT mice treated with vehicle or the PPAR beta/delta agonist GW501516, and C2C12 myotubes treated with the ER stressor tunicamycin or different activators or inhibitors were used.ResultsPpard-/- mice displayed reduced InsR beta protein levels in their skeletal muscle compared to wild-type (WT) mice, while the PPAR beta/delta agonist GW501516 increased its levels in WT mice. Co-incubation of tunicamycin-exposed C2C12 myotubes with GW501516 partially reversed the decrease in InsR beta protein levels, attenuating both ER stress and the increase in lysosomal activity. In addition, the protein levels of the tyrosine kinase ephrin receptor B4 (EphB4), which binds to the InsR beta and facilitates its endocytosis and degradation in lysosomes, were increased in the skeletal muscle of Ppard-/- mice, with GW501516 reducing its levels in the skeletal muscle of WT mice.ConclusionsOverall, these findings reveal that PPAR beta/delta activation increases InsR beta levels by alleviating ER stress and lysosomal degradation.

Filiaciones:
Wang JR:
 Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Food Sciences, Institute of Biomedicine of the University of Barcelona (IBUB), University of Barcelona, Barcelona, 08028, Spain

 Spanish Biomedical Research Center in Diabetes and Associated Metabolic Diseases (CIBERDEM)-Instituto de Salud Carlos III, Madrid, 28029, Spain

 Pediatric Research Institute-Hospital Sant Joan de Déu, Esplugues de Llobregat, Spain

Jurado-Aguilar J:
 Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Food Sciences, Institute of Biomedicine of the University of Barcelona (IBUB), University of Barcelona, Barcelona, 08028, Spain

 Spanish Biomedical Research Center in Diabetes and Associated Metabolic Diseases (CIBERDEM)-Instituto de Salud Carlos III, Madrid, 28029, Spain

 Pediatric Research Institute-Hospital Sant Joan de Déu, Esplugues de Llobregat, Spain

Barroso E:
 Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Food Sciences, Institute of Biomedicine of the University of Barcelona (IBUB), University of Barcelona, Barcelona, 08028, Spain

 Spanish Biomedical Research Center in Diabetes and Associated Metabolic Diseases (CIBERDEM)-Instituto de Salud Carlos III, Madrid, 28029, Spain

 Pediatric Research Institute-Hospital Sant Joan de Déu, Esplugues de Llobregat, Spain

Rodríguez-Calvo R:
 Spanish Biomedical Research Center in Diabetes and Associated Metabolic Diseases (CIBERDEM)-Instituto de Salud Carlos III, Madrid, 28029, Spain

 Vascular Medicine and Metabolism Unit, Research Unit on Lipids and Atherosclerosis, "Sant Joan" University Hospital, Universitat Rovira i Virgili, Pere Virgili Health Research Institute (IISPV), Reus, 43201, Spain

Camins A:
 Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Food Sciences, Institute of Biomedicine of the University of Barcelona (IBUB), University of Barcelona, Barcelona, 08028, Spain

 Biomedical Research Networking Centre in Neurodegenerative Diseases (CIBERNED), Madrid, Spain

 Institute of Neuroscience, University of Barcelona, Barcelona, Spain

Wahli W:
 Center for Integrative Genomics, University of Lausanne, Lausanne, CH-1015, Switzerland

 INRAE ToxAlim, UMR1331, Chemin de Tournefeuille, Toulouse Cedex 3, F-31027, France

Palomer FX:
 Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Food Sciences, Institute of Biomedicine of the University of Barcelona (IBUB), University of Barcelona, Barcelona, 08028, Spain

 Spanish Biomedical Research Center in Diabetes and Associated Metabolic Diseases (CIBERDEM)-Instituto de Salud Carlos III, Madrid, 28029, Spain

 Pediatric Research Institute-Hospital Sant Joan de Déu, Esplugues de Llobregat, Spain

Vazquez M:
 Department of Pharmacology, Toxicology and Therapeutic Chemistry, Faculty of Pharmacy and Food Sciences, Institute of Biomedicine of the University of Barcelona (IBUB), University of Barcelona, Barcelona, 08028, Spain

 Spanish Biomedical Research Center in Diabetes and Associated Metabolic Diseases (CIBERDEM)-Instituto de Salud Carlos III, Madrid, 28029, Spain

 Pediatric Research Institute-Hospital Sant Joan de Déu, Esplugues de Llobregat, Spain

 Unitat de Farmacologia, Facultat de Farmàcia i Ciències de l'Alimentació, Av. Joan XXIII 27-31, Barcelona, E-08028, Spain
ISSN: 1478811X





Cell Communication and Signaling
Editorial
BMC, CAMPUS, 4 CRINAN ST, LONDON N1 9XW, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 22 Número: 1
Páginas: 595-595
WOS Id: 001380104800002
ID de PubMed: 39696437
imagen Green Submitted, gold

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