The natural history of variable subtypes in pediatric-onset TUBB4A-related leukodystrophy.


Por: Gavazzi F, Charsar B, Hamilton E, Erler JA, Patel V, Woidill S, Sevagamoorthy A, Helman G, Schmidt J, Pizzino A, Muirhead K, Takanohashi A, Bonkowsky JL, Meyerhoffer K, Simons C, Doi H, Satoko M, Matsumoto N, Delgado MR, Sanchez-Castillo M, Wang J, de Carvalho DR, Tournev I, Chamova T, Jordanova A, Clegg NJ, Nicita F, Bertini E, Teng M, Williams D, Tonduti D, Houlden H, Stellingwerff M, Wassmer E, Garcia-Cazorla A, Bernard G, Mirchi A, Toutounchi H, Wolf NI, van der Knaap MS, Shults J, Adang LA and Vanderver AL

Publicada: 1 mar 2025 Ahead of Print: 1 feb 2025
Resumen:
We establish the natural history of pediatric-onset TUBB4A-related leukodystrophy to improve clinical trial readiness through a medical record-based longitudinal study. An international cohort of 216 individuals with pediatric-onset TUBB4A-related leukodystrophy was included. Demographic information and medical events were extracted from medical records or publications. Retrospective scores (Gross Motor Function - Metachromatic Leukodystrophy [GMFC-MLD] and Communication Function Classification System [CFCS]) were applied to assess function. Survival analysis distinguished differences in longitudinal neurocognitive function and time to event outcomes between subtypes. A decision tree predicted independent ambulation from early motor milestones. Genotype (p.Asp249Asn vs non-p.Asp249Asn) and independent sitting by age 9 months predicted ambulation by 3 years, and stratification into three subgroups: early-infantile (non- sitting by 9 months), late-infantile (normal early milestones without the common p.Asp249Asn mutation), and a cohort of p.Asp249Asn late-infantile onset individuals. Median age at symptom onset was 0.71 years (interquartile range: [0.33, 1.50]). Common symptoms at onset include delayed development and tone abnormalities (n = 125, 66.5 % and n = 77, 43.0 %). The most common medical complications included scoliosis (N = 51/142), hip dislocation (N = 30/101), and seizures (N = 51/163). The early-infantile more severely affected cohort had a greater prevalence of G-tube placement, scoliosis, and seizure compared to the late-infantile form (p < 0.01). Peak motor and communication abilities were comparable between the p.Asp249Asn and the late infantile cohorts. Despite the acquisition of early milestones, individuals with p.Asp249Asn showed a more rapid decline of functional abilities compared to other late infantile forms (log-rank p = 0.0002). Better understanding of TUBB4A-related leukodystrophy subtypes will improve clinical care, allow targeted preventive interventions, and permit disease stratification for future disease-modifying clinical trials.

Filiaciones:
Gavazzi F:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Charsar B:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Hamilton E:
 Department of Child Neurology, Amsterdam Leukodystrophy Center, Emma Children's Hospital, Amsterdam University Medical Centers, Netherlands

Erler JA:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Patel V:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Woidill S:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Sevagamoorthy A:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Helman G:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Schmidt J:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Pizzino A:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Muirhead K:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Takanohashi A:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

Bonkowsky JL:
 Division of Pediatric Neurology, Department of Pediatrics, University of Utah School of Medicine, Salt Lake City, UT, USA

 Center for Personalized Medicine, Primary Children's Hospital, Salt Lake City, UT, USA

Meyerhoffer K:
 Division of Pediatric Neurology, Department of Pediatrics, University of Utah School of Medicine, Salt Lake City, UT, USA

Simons C:
 Murdoch Childrens Research Institute, Melbourne, Australia

Doi H:
 Department of Neurology and Stroke Medicine, Yokohama City University Graduate School of Medicine, Yokohama, Japan

Satoko M:
 Department of Human Genetics, Yokoama City University, Yokohama, Japan

Matsumoto N:
 Department of Human Genetics, Yokoama City University, Yokohama, Japan

Delgado MR:
 Department of Neurology, University of Texas Southwestern Medical Center, USA

Sanchez-Castillo M:
 TGen's Center for Rare Childhood Disorders, Phoenix, AZ, USA

Wang J:
 Department of Pediatrics, Peking University First Hospital, Beijing, China

de Carvalho DR:
 Genetic Unit, SARAH Rehabilitation Hospital, Brasilia, Brazil

Tournev I:
 Clinic of Nervous Diseases, University Hospital Aleksandrovska, Department of Neurology, Medical University-Sofia, Bulgaria

 Department of Cognitive Science and Psychology, New Bulgarian University, Bulgaria

Chamova T:
 Department of Neurology, University Hospital Alexandrovska, Medical University Sofia, Sofia, Bulgaria

Jordanova A:
 Molecular Medicine Center, Department of Medical Chemistry and Biochemistry, Medical University - Sofia, Sofia, Bulgaria

 Molecular Neurogenomics Group, Center for Molecular Neurology, VIB, Antwerpen, Belgium

 Molecular Neurogenomics Group, Center for Molecular Neurology, Department of Biomedical Sciences, University of Antwerp, Antwerpen, Belgium

Clegg NJ:
 Division of Clinical Research, Department of Research, Scottish Rite for Children, Dallas, TX, USA

Nicita F:
 Unit of Neuromuscular and Neurodegenerative Diseases, Department of Neuroscience and Neurorehabilitation, IRCCS Bambino Gesù Children's Hospital, Rome, Italy

Bertini E:
 Research Unit of Neuromuscular and Neurodegenerative Diseases, Bambino Gesu' Children's Hospital, IRCCS, Rome, Italy

Teng M:
 Synaptixbio Ltd, Fermi Avenue, Harwell, UK

Williams D:
 Synaptixbio Ltd, Fermi Avenue, Harwell, UK

Tonduti D:
 Unit of Pediatric Neurology, C.O.A.L.A (Center for Diagnosis and Treatment of Leukodystrophies), V. Buzzi Children's Hospital, Milan, Italy

 Department of Biomedical and Clinical Sciences, L. Sacco University Hospital, Università degli Studi di Milano, Milan, Italy

Houlden H:
 Department of Clinical and Movement Neurosciences, UCL Queen Square Institute of Neurology, University College London, London, UK

Stellingwerff M:
 Department of Pediatric Surgery, Vrije Universiteit, Amsterdam, Netherlands

Wassmer E:
 Neurology Department, Birmingham Children's Hospital, Institute of Health and Neurodevelopment, Aston University, Birmingham, UK

Garcia-Cazorla A:
 Institut de Recerca Sant Joan de Déu, Barcelona, Spain

 Neurology Department, Neurometabolic Unit and Synaptic Metabolism Lab, Institut Pediàtric de Recerca, Hospital Sant Joan de Déu, metabERN and CIBERER-ISCIII, Barcelona, Spain

Bernard G:
 Departments of Neurology and Neurosurgery, Pediatrics and Human Genetics, McGill University, Montreal, Quebec, Canada

 Child Health and Human Development Program, Research Institute of the McGill University Health Centre, Montreal, Quebec, Canada

 Department of Specialized Medicine, Division of Medical Genetics, McGill University Health Centre, Montreal, Quebec, Canada

Mirchi A:
 Departments of Neurology and Neurosurgery, Pediatrics and Human Genetics, McGill University, Montreal, Quebec, Canada

Toutounchi H:
 Departments of Neurology and Neurosurgery, Pediatrics and Human Genetics, McGill University, Montreal, Quebec, Canada

Wolf NI:
 Department of Child Neurology, Amsterdam Leukodystrophy Center, Emma Children's Hospital, Amsterdam University Medical Centers, and Amsterdam Neuroscience, Cellular & Molecular Mechanisms, Vrije Universiteit, Amsterdam, Netherlands

van der Knaap MS:
 Department of Child Neurology, Amsterdam Leukodystrophy Center, Emma Children's Hospital, Amsterdam University Medical Centers, and Amsterdam Neuroscience, Cellular & Molecular Mechanisms, Vrije Universiteit, Amsterdam, Netherlands

Shults J:
 Department of Pediatrics, Children's Hospital of Philadelphia, Philadelphia, PA, USA

 Department of Biostatistics, Epidemiology and Informatics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA

Adang LA:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

 Department of Neurology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA

Vanderver AL:
 Neurology Department, Children's Hospital of Philadelphia, Philadelphia, PA, USA

 Department of Neurology, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA
ISSN: 10967192





MOLECULAR GENETICS AND METABOLISM
Editorial
ACADEMIC PRESS INC ELSEVIER SCIENCE, 525 B ST, STE 1900, SAN DIEGO, CA 92101-4495, Estados Unidos America
Tipo de documento: Article
Volumen: 144 Número: 3
Páginas: 109048-109048
WOS Id: 001428601500001
ID de PubMed: 39951964
imagen Green Submitted, Green Published

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