A novel human pluripotent stem cell gene activation system identifies IGFBP2 as a mediator in the production of haematopoietic progenitors in vitro.


Por: Petazzi, P, Ventura, T, Luongo, FP, McClafferty, H, May, A, Taylor, HA, Shipston, MJ, Romanò, N, Forrester, LM, Menendez, P and Fidanza, A

Publicada: 23 dic 2024 Ahead of Print: 23 dic 2024
Resumen:
A major challenge in the stem cell biology field is the ability to produce fully functional cells from induced pluripotent stem cells (iPSCs) that are a valuable resource for cell therapy, drug screening, and disease modelling. Here, we developed a novel inducible CRISPR-mediated activation strategy (iCRISPRa) to drive the expression of multiple endogenous transcription factors (TFs) important for in vitro cell fate and differentiation of iPSCs to haematopoietic progenitor cells. This work has identified a key role for IGFBP2 in developing haematopoietic progenitors. We first identified nine candidate TFs that we predicted to be involved in blood cell emergence during development, then generated tagged gRNAs directed to the transcriptional start site of these TFs that could also be detected during single-cell RNA sequencing (scRNAseq). iCRISPRa activation of these endogenous TFs resulted in a significant expansion of arterial-fated endothelial cells expressing high levels of IGFBP2, and our analysis indicated that IGFBP2 is involved in the remodelling of metabolic activity during in vitro endothelial to haematopoietic transition. As well as providing fundamental new insights into the mechanisms of haematopoietic differentiation, the broader applicability of iCRISPRa provides a valuable tool for studying dynamic processes in development and for recapitulating abnormal phenotypes characterised by ectopic activation of specific endogenous gene expression in a wide range of systems.

Filiaciones:
Petazzi, P:
 Josep Carreras Leukemia Res Inst, Barcelona, Spain

Ventura, T:
 Univ Edinburgh, Inst Regenerat & Repair, Ctr Regenerat Med, Edinburgh, Midlothian, Scotland

Luongo, FP:
 Univ Edinburgh, Inst Regenerat & Repair, Ctr Regenerat Med, Edinburgh, Midlothian, Scotland

McClafferty, H:
 Univ Edinburgh, Edinburgh Med Sch, Ctr Discovery Brain Sci, Biomed Sci, Edinburgh, Midlothian, Scotland

May, A:
 Univ Edinburgh, Inst Regenerat & Repair, Ctr Regenerat Med, Edinburgh, Midlothian, Scotland

Taylor, HA:
 Univ Edinburgh, Inst Regenerat & Repair, Ctr Regenerat Med, Edinburgh, Midlothian, Scotland

Shipston, MJ:
 Univ Edinburgh, Edinburgh Med Sch, Ctr Discovery Brain Sci, Biomed Sci, Edinburgh, Midlothian, Scotland

 Zhejiang Univ, Zhejiang Univ Univ Edinburgh Joint Inst, Sch Med, Haining, Peoples R China

Romanò, N:
 Univ Edinburgh, Edinburgh Med Sch, Ctr Discovery Brain Sci, Biomed Sci, Edinburgh, Midlothian, Scotland

 Zhejiang Univ, Zhejiang Univ Univ Edinburgh Joint Inst, Sch Med, Haining, Peoples R China

Forrester, LM:
 Univ Edinburgh, Inst Regenerat & Repair, Ctr Regenerat Med, Edinburgh, Midlothian, Scotland

:
 Josep Carreras Leukemia Res Inst, Barcelona, Spain

 CIBER ONC, ISCIII, Barcelona, Spain

 ICREA, Barcelona, Spain

 Univ Barcelona, Sch Med, Dept Biomedicine, Barcelona, Spain

 Inst Recerca St Joan de Deu PCCB SJD, Pediat Canc Ctr Barcelona, Barcelona, Spain

Fidanza, A:
 Univ Edinburgh, Inst Regenerat & Repair, Ctr Regenerat Med, Edinburgh, Midlothian, Scotland

 Univ Edinburgh, Edinburgh Med Sch, Biomed Sci, Edinburgh, Midlothian, Scotland
ISSN: 2050084X





eLife
Editorial
eLIFE SCIENCES PUBL LTD, 95 Regent Street, CAMBRIDGE CB2 1AW, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 13 Número:
Páginas:
WOS Id: 001394474500001
ID de PubMed: 39714446
imagen Green Submitted, gold

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